Upon completion of the planned program, units receive an ASGE ‘Certificate of Recognition’ for promoting quality in endoscopy. This symbol can be displayed to allow individuals and referring physicians know that the endoscopy unit is dedicated to delivering high-quality endoscopic care and has received specialized training around these principles. New ASGE Endoscopy Device Recognition System Honorees Beverly Hospital Endoscopy Division, Beverly, MA Bulloch County Endoscopy Middle, LLC, Statesboro, GA Butler Ambulatory Surgery Center, Butler, PA Chang Ho Suk, MD, Flushing, NY Charlottesville Gastroenterology, Charlottesville, VA Davis Memorial Endoscopy Middle, Elkins, WV Digestive Treatment and Diagnostic Middle, Pittsburgh, PA Digestive Wellness Experts, PA Thomasville, Thomasville, NC Drexel Centers for Digestive Health, Philadelphia, PA Endoscopy Center of Dollars County, Newtown, PA Endoscopy Middle of Long Island, Backyard City, NY Gastro One, G.I.Choi, M. Bodria, Y. Liu, P.L. Weng, V.J. Lozanovski, M. Verbitsky, F. Lugani, R. Sterken, N. Paragas, G. Caridi, A. Carrea, M. Dagnino, A. Materna-Kiryluk, G. Santamaria, C. Murtas, N. Ristoska-Bojkovska, C. Izzi, N. Kacak, B. Bianco, S. Giberti, M. Gigante, G. Piaggio, L. Gesualdo, D. Kosuljandic Vukic, K. Vukojevic, M. Saraga-Babic, M. Saraga, Z. Gucev, L. Allegri, A. Latos-Bielenska, D. Casu, M. Condition, F. Scolari, R. Ravazzolo, K. Kiryluk, Q. Al-Awqati, V.D. D’Agati, I.A. Drummond, V. Tasic, R.P. Lifton, G.M. Ghiggeri, and A.G. Gharavi: Mutations in DSTYK and Dominant URINARY SYSTEM Malformations Congenital malformations of the kidney and urinary system contribute to 23 percent of birth defects1,2 and take into account 40 to 50 percent of pediatric cases and 7 percent of adult situations of end-stage renal disease worldwide.3,4 These disorders are heterogeneous and encompass an array of anatomical defects genetically, such as for example renal agenesis, renal hypodysplasia, ureteropelvic junction obstruction, or vesicoureteral reflux.5 Mutations in genes that trigger syndromic disorders, such as PAX2 and HNF1B mutations, are detected in only 5 to 10 percent of cases.6,7 Familial forms of nonsyndromic disease have been reported, further supporting genetic determination8,9; however, owing to locus heterogeneity and little pedigree size, the genetic reason behind most sporadic or familial cases remains unknown.